A patient born in 1958. Clinical diagnosis - Nodular toxic goiter. Accompanying - severe anemia grade III. Complaints include neck pain (in the thyroid projection), a sensation of a foreign body, shortness of breath, loss of consciousness, pale skin, hot flashes at bedtime, and a subcutaneous hardening on the left foot the size of a nut. Symptoms began half a year ago. The patient was examined at the Institute of Endocrinology. During the examination, severe anemia was detected. Before coming to us, she received treatment at the blood transfusion institute. After the treatment, her condition slightly improved, but complaints of neck pain persisted. An endocrinologist recommended surgery.
Past illnesses: ARVI, catarrhal angina, periodontitis, anemia. No harmful habits. No family history of the disease.
On examination: General condition relatively satisfactory. Skin and visible mucous membranes clean, pale. Throat calm. Peripheral lymph nodes not enlarged. Musculoskeletal system - there is spinal deformity (scoliosis). Visually: thyroid diffusely enlarged, grade III. Consistency is firm-elastic. In the lungs - vesicular breathing. Heart sounds clear, slight systematic murmur at the apex. BP - 90/60.
Abdomen soft, non-painful. Liver and spleen not palpable. Bowel movements normal.
Laboratory data: Thyroid function test - thyroid image deformed due to the absence of the upper pole and middle zone of the right lobe. Left lobe and isthmus enlarged. Central thyroid function within normal limits. Conclusion: extensive "cold" defect of accumulation, replacing the volume of the upper pole and middle zone of the right lobe. Compensatory hypertrophy of the left lobe and isthmus.
Ultrasound: from January 12, 1998, thyroid gland - right lobe 39x25 mm, where two nodules are visible - 16x11 mm, and 17x15 mm. Left lobe - 35x25 mm, one nodule - 17x11 mm. In both lobes, smaller nodules of 5 and 7 mm are visible. Conclusion: Nodular goiter.
Blood test: from October 16, 1997, after therapy - Hb - 107 g/L, erythrocytes - 3.5x10^12/L, CI - 0.8. Reticulocytes 56% (on the background of A12 injections), thrombocytes - 112% - 392x10^9/L, neutrophils - 60.0, eosinophils - 7.0, lymphocytes - 28.0, ESR - 13 mm/h, macrocytosis.
EAF diagnosis results: Readings above normal on meridians: Lymphatic system KT 1-78/74, 3-70. Lungs point 10 - 80/82. Nervous system point 1 - 70, 1a - 78, KT - 78, 3 - 78, 3a - 76. Blood vessels 8c - 72, 8a - 70, 8 - 74. Allergy 1 - 72, KT - 74. Epithelium and parenchyma 1 - 78, 1.1 - 72, 2 - 80.
Treatment: Conducted according to the general regime. When local emitters were placed on the thyroid area, normalization of readings was not observed, neither on the thyroid point nor on points of other organs. However, when the GI emitter was applied to the lung area, readings of all organs (including the thyroid) moved towards normal.
The results of the emitter testing can be commented on as follows. The inflammatory process in the lung area forces the body into anaerobic energy exchange, which requires additional energy supplied by intensified protein anabolism and catabolism. This, in turn, leads to adrenal hormone insufficiency due to high consumption, which was compensated by increased functional activity of the thyroid gland (since thyroid hormones can partially substitute for adrenal hormones), leading to nodule formation.
In line with our assumptions about the causes and mechanisms of this disease's development, treatment focused on the lung area and increasing the functional activity of the adrenal glands, gallbladder (as a source of free cholesterol when exposed to appropriate emitters), and intestines, which are primarily responsible for delivering necessary energy and building materials.
After a month-long therapy course, the patient's condition significantly improved; her ability to work increased, weakness is rare. Hb increased from 107 g/L to 130 g/L. No evening hot flashes.
Ultrasound of the thyroid gland from February 18, 1998: Right lobe 47x36x32 mm, multiple nodules of 4-10 ml relatively homogeneous. Left lobe 56x28x45 mm, parenchyma unevenly dense, multiple nodules up to 10 mm in structure. Isthmus 15 mm thick, nodule d - 8 mm in the isthmus parenchyma.
Ultrasound from March 14, 1998: Thyroid gland, right lobe 42x28x32 mm, multiple nodules from 4 to 9 mm in diameter. Left lobe 36x28x41 mm, two large nodules - 11x9 mm, 12x8 mm, and smaller ones up to 8 mm. Isthmus 16 mm thick, nodule d - 6 mm.
Past illnesses: ARVI, catarrhal angina, periodontitis, anemia. No harmful habits. No family history of the disease.
On examination: General condition relatively satisfactory. Skin and visible mucous membranes clean, pale. Throat calm. Peripheral lymph nodes not enlarged. Musculoskeletal system - there is spinal deformity (scoliosis). Visually: thyroid diffusely enlarged, grade III. Consistency is firm-elastic. In the lungs - vesicular breathing. Heart sounds clear, slight systematic murmur at the apex. BP - 90/60.
Abdomen soft, non-painful. Liver and spleen not palpable. Bowel movements normal.
Laboratory data: Thyroid function test - thyroid image deformed due to the absence of the upper pole and middle zone of the right lobe. Left lobe and isthmus enlarged. Central thyroid function within normal limits. Conclusion: extensive "cold" defect of accumulation, replacing the volume of the upper pole and middle zone of the right lobe. Compensatory hypertrophy of the left lobe and isthmus.
Ultrasound: from January 12, 1998, thyroid gland - right lobe 39x25 mm, where two nodules are visible - 16x11 mm, and 17x15 mm. Left lobe - 35x25 mm, one nodule - 17x11 mm. In both lobes, smaller nodules of 5 and 7 mm are visible. Conclusion: Nodular goiter.
Blood test: from October 16, 1997, after therapy - Hb - 107 g/L, erythrocytes - 3.5x10^12/L, CI - 0.8. Reticulocytes 56% (on the background of A12 injections), thrombocytes - 112% - 392x10^9/L, neutrophils - 60.0, eosinophils - 7.0, lymphocytes - 28.0, ESR - 13 mm/h, macrocytosis.
EAF diagnosis results: Readings above normal on meridians: Lymphatic system KT 1-78/74, 3-70. Lungs point 10 - 80/82. Nervous system point 1 - 70, 1a - 78, KT - 78, 3 - 78, 3a - 76. Blood vessels 8c - 72, 8a - 70, 8 - 74. Allergy 1 - 72, KT - 74. Epithelium and parenchyma 1 - 78, 1.1 - 72, 2 - 80.
Treatment: Conducted according to the general regime. When local emitters were placed on the thyroid area, normalization of readings was not observed, neither on the thyroid point nor on points of other organs. However, when the GI emitter was applied to the lung area, readings of all organs (including the thyroid) moved towards normal.
The results of the emitter testing can be commented on as follows. The inflammatory process in the lung area forces the body into anaerobic energy exchange, which requires additional energy supplied by intensified protein anabolism and catabolism. This, in turn, leads to adrenal hormone insufficiency due to high consumption, which was compensated by increased functional activity of the thyroid gland (since thyroid hormones can partially substitute for adrenal hormones), leading to nodule formation.
In line with our assumptions about the causes and mechanisms of this disease's development, treatment focused on the lung area and increasing the functional activity of the adrenal glands, gallbladder (as a source of free cholesterol when exposed to appropriate emitters), and intestines, which are primarily responsible for delivering necessary energy and building materials.
After a month-long therapy course, the patient's condition significantly improved; her ability to work increased, weakness is rare. Hb increased from 107 g/L to 130 g/L. No evening hot flashes.
Ultrasound of the thyroid gland from February 18, 1998: Right lobe 47x36x32 mm, multiple nodules of 4-10 ml relatively homogeneous. Left lobe 56x28x45 mm, parenchyma unevenly dense, multiple nodules up to 10 mm in structure. Isthmus 15 mm thick, nodule d - 8 mm in the isthmus parenchyma.
Ultrasound from March 14, 1998: Thyroid gland, right lobe 42x28x32 mm, multiple nodules from 4 to 9 mm in diameter. Left lobe 36x28x41 mm, two large nodules - 11x9 mm, 12x8 mm, and smaller ones up to 8 mm. Isthmus 16 mm thick, nodule d - 6 mm.